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Download the version of MSXML you need here. Some MSXML. Microsoft Office 2010 Setup.The following description is provided to assist the understanding of the reader. None of the information provided or references cited is admitted to be prior art to the present description.
Coronary heart disease is a leading cause of death in the United States. Atherosclerosis, a disease involving the accumulation of cholesterol and other lipids in the artery walls, is the most common disease resulting in coronary heart disease. Increasingly, early detection of atherosclerosis is desired. A number of biochemical markers have been investigated to detect atherosclerosis including indicators of inflammation, such as C-reactive protein. Biochemical markers of inflammation, however, are not without limitations. For instance, a significant increase in C-reactive protein levels in serum may indicate an inflammatory condition, but it may also be indicative of another disease, e.g., chronic infection.
Another area of active research for atherosclerosis detection is the field of molecular imaging. Molecular imaging refers to the visualization of an agent with a specific binding affinity to a molecular target of interest using conventional imaging techniques, such as magnetic resonance imaging (MRI), computed tomography (CT), ultrasound, etc. The imaging technique may involve the detection of a molecular binding event using a probe of known structure and function. Alternatively, imaging techniques are based on monitoring a real-time change in one or more physical properties of a molecule or a region of interest during a specific molecular binding event.
Many detectable molecular binding events are characterized by biochemical processes that occur at the molecular level and depend upon the nucleophilicity of the molecules involved. For example, nitric oxide (NO) is a reactive free radical gas which is able to diffuse to adjacent molecules through an unstirred aqueous layer, where it reacts with electron-rich side groups on proteins and DNA. NO is typically generated in mammalian cells through the enzymatic action of nitric oxide synthase (NOS). Binding of an NO molecule to an electron-rich group on a molecule can lead to the formation of a nitroso functional group.

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