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The development of carbapenem-resistance among some strains of Klebsiella pneumoniae and other Enterobacteriaceae is now an important, and difficult to control problem. The mechanisms of carbapenem-resistance are complex and primarily involve the acquisition of carbapenem-hydrolysing beta-lactamases, which result in the active-site modification of non-carbapenem beta-lactamases. Metalloenzymes, such as mecA and bla(mec) genes, are other important determinants of carbapenem-resistance and are encoded by class 1 integrons. The increase in the number of these enzymes and the loss of the chromosomal mecA gene are associated with an increase in carbapenem resistance. The success of bacterial populations can be determined by a variety of factors, including the presence of plasmids, the mechanisms of plasmid carriage and the frequency of transfer of these factors to other bacteria. It appears that carbapenem resistance may become a major factor in hospital and community bacteriology. To date, the impact of this resistance on therapeutic options is unclear. The potential impact of carbapenem resistance on the choice of hospital infections and, in particular, on meningitis, requires careful consideration. In the future, the development of appropriate measures to prevent the emergence of carbapenem resistance are needed.The Galloway & Tobin Family History

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