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Aberrant expression of nuclear factor of activated T-cells 5 in multiple endocrine neoplasia type 1 suggests its involvement in a micro-destruction of the parathyroid gland.
Multiple endocrine neoplasia type 1 (MEN1) syndrome is characterized by the onset of various endocrine tumors, including nonfunctioning (NFP) and/or hyperfunctioning (HFP) parathyroid adenomas and pancreatic neuroendocrine tumors (NETs). To find novel genes involved in the tumorous process of the MEN1 syndrome, we performed suppression subtractive hybridization analysis (SSH) to isolate differentially expressed genes from a MEN1-associated parathyroid adenoma and a MEN1-derived NET. One of the upregulated genes (BMP-1) was cloned from a MEN1-associated parathyroid adenoma and compared with that of the normal parathyroid gland. BMP-1 messenger RNA expression was examined by Northern blot analysis and in situ hybridization in 11 specimens of MEN1-associated parathyroid adenomas and 4 normal parathyroid glands. Subcellular localization of BMP-1 was investigated by immunohistochemistry in 5 normal parathyroid glands and 12 hyperplastic and neoplastic parathyroid glands obtained from MEN1 patients. BMP-1 mRNA was expressed at a higher level in MEN1-associated parathyroid adenomas than in normal parathyroid glands by Northern blot analysis. In situ hybridization showed strong BMP-1 mRNA expression in the nuclei of the tumor cells of MEN1-associated parathyroid adenomas. Immunohistochemical analysis showed BMP-1 expression in the nuclei of hyperplastic parathyroid gland, adenoma, and NET as well as in other endocrine cells. On the other hand, BMP-1 was almost absent in the cytoplasm of normal parathyroid cells. These results suggest that aberrant BMP-1 expression in multiple endocrine neoplasms may play an important role in the microdestruction of the parathyroid gland.Q:

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